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showed a small, albeit statistically significant, increase in HbA1c in one long-term study, while no effect was observed in another long-term study and no significant effects were noted in short-term studies among patients with non-diabetic peripheral neuropathic pain [164]

Independent groups have observed consistent patterns across the published research record, providing cross-laboratory replication for the core mechanism findings documented in the foundational papers cited above

Demyelination and remyelination in anatomically distinct regions of the corpus callosum following cuprizone intoxication